EBV and P. falciparum influence the risk of IG::MYC translocations, which are a hallmark feature of BL [1], by upregulating activation-induced cytidine deaminase (AICDA) [9], a mutator enzyme involved in class switch recombination and somatic hypermutation in B-cells [10, 11]. Here, AICDA is linked to Burkitt lymphoma.