As it was mentioned earlier, in our experiment, only the highest CBG concentration (10 μM) diminished the level of CD36 and consequently the accumulation of lipids in the PA overload conditions (Figure 8), which may be advantageous in the states of FA oversupply (e.g., in obesity) as it protects against the excessive FA uptake into the cardiomyocytes and further lipotoxicity development [35]. The gene discussed is CD36; the disease is obesity due to melanocortin 4 receptor deficiency.