Within the BLA, specific interneuron subtypes, such as parvalbumin (PV+), somatostatin (SOM+), and cholecystokinin (CCK+) interneurons, modulate amygdala output and anxiety-like behaviors, and facilitation of endocannabinoid signaling, respectively (Urakawa et al., 2013; Fuchs et al., 2017; Trouche et al., 2013). The gene discussed is CCK; the disease is Anxiety.