Correspondingly, specific DEGs included upregulations of typical pro-inflammatory molecules involved in vascular inflammation, such as C-C motif chemokine ligand 2/monocyte chemoattractant protein 1 (CCL2/MCP-1), C-X3-C motif chemokine ligand 1 (Fractalkine/CX3CL1), intercellular adhesion molecule 1 (ICAM1), vascular cell adhesion molecule 1 (VCAM1), selectin E (SELE). The gene discussed is CCL2; the disease is inflammatory response.