We also report 147 colocalized AD‐GWAS and cis‐edQTLs signals relating to 48 likely causal genes comprising CLU (rs7982, rs1532278), BIN1 (rs2276582, rs3768863), GRIN3B (rs10417824, rs1058603), NYAP1 (rs12539172), DGKQ (rs4690197, rs3733347), and CLPTM1 (rs204468), mainly implicated in tau protein binding, amyloid‐β regulation, cellular morphogenesis, and immune response pathway, suggesting possible role of epitranscriptomic mechanism in the disease pathology. Here, CLPTM1 is linked to Alzheimer disease.