Chronic myeloid leukemia (CML) is a hematologic malignancy characterized by a clonal proliferation of myeloid cells due to the presence of the BCR-ABL1 fusion gene, a consequence of the Philadelphia chromosome translocation t (9;22) (q34; q11) [1]. This evidence concerns the gene ABL1 and chronic myelogenous leukemia, BCR-ABL1 positive.