Although the generation of Mo-AMs appears to not be affected by Dicer deletion during BLM-PF, we cannot rule out the possibility that some individual miRNAs may still play a role in Mo-AM production under fibrosis disease conditions, and that deficiency of these miRNAs in DicerKO mice may offset their promoting or inhibitory functions in the generation of Mo-AMs from BM during fibrogenesis. This evidence concerns the gene DICER1 and fibrosis.