STING1 and Alzheimer disease: Thirdly, while the mechanisms underlying microglial senescence‐induced cognitive dysfunction were not investigated in this study, evidence suggests that the cGAS‐STING pathway is a key driver of senescence‐associated neuroinflammation and neurodegeneration in AD models (Carling et al. 2024; Yuan et al. 2025), a mechanism that warrants investigation in our future studies.