Since we found that increased eEF2K expression drives cell proliferation, invasion, and tumor growth, we next examined the interplay between eEF2K and TAM-derived factors such as MCP-1, a key chemokine involved in the recruitment and tumoral infiltration of monocytes [24] as well as monocyte-macrophage differentiation [25, 26]. The gene discussed is EEF2K; the disease is neoplasm.