EGFR and neoplasm: We show that tumour epithelial cells with high EGFR expression featured – (a) significant down‐regulation of (a.1) antigen‐processing and presentation (HLA‐A, HLA‐B, HLA‐DRA, B2M, etc.), (a.2) immune‐stimulating genes (CXCL17, LCN2 and SAA1), and (b) significant up‐regulation of (b.1) pro‐angiogenic factors VEGFB and FGFBP1, (b.2) glutathione biosynthesis related gene GPX2, and its interacting partners AKR1C1, AKR1C2 and AKR1C3 (Figure 3F).