Several pathways related to proliferation (Myc target and G2/M checkpoint), fibrosis (TGF‐β signalling), cell death (p53 and apoptosis) and cellular metabolism (cholesterol homeostasis, adipogenesis and oxidative phosphorylation) were changed in the 16‐week tumours when compared with the 8‐week precancer lesions (Figure 2E). The gene discussed is MYC; the disease is neoplasm.