To enhance specificity, gene expression profiles of rheumatoid arthritis (RA)—commonly used as a positive control for systemic inflammation in biomedical research due to shared Th1/Th17 response pathways cytokine profiles (IL-17, TNF-α, and IFN-γ) with IBD (7–9)—were also examined. This evidence concerns the gene IL17A and rheumatoid arthritis.