Upon RL (660 nm) exposure, FnBphP interacts with LDB3, enabling reconstitution of a functional complex that can bind to the P5 × UAS promoter via the Gal4 DBD domain to initiate expression of the immune adjuvant IFN‐β, which has been shown as attractive candidate adjuvant for cancer vaccination.[42] Conversely, upon FRL (780 nm) exposure, the FnBphP will dissociate from LDB3, which can inactivate IFN‐β expression (Figure 2A). This evidence concerns the gene LGALS4 and cancer.