To further delve into the prospective role of circACTN4 through the circACTN4/MYC/AcH4 axis, several assays were conducted in BC cells transfected with circACTN4 overexpressing plasmid or si-circACTN4 and cotransfection of circACTN4 and si-MYC or si-circACTN4 and OE-MYC. This evidence concerns the gene MYC and breast cancer.