Both inflammation and lipid metabolism are known to contribute to the likelihood of sarcopenia (29, 30), with elevated pro-inflammatory markers, such as Tumor Necrosis Factor-alpha (TNF-α), Interleukin-6(IL-6), and C-Reactive Protein (CRP), being observed in both sarcopenia and dyslipidemia, suggesting shared pathogenic mechanisms (31–33). The gene discussed is TNF; the disease is sarcopenia.