The peroxisome proliferator-activated receptor (PPAR) signaling pathway, which plays a crucial role as a link between lipid signaling and inflammatory responses, has garnered widespread attentions for its dysfunction in the liver of IUGR piglets, characterized by the reduced mRNA levels of PPARA and the elevated mRNA levels of PPARG and CD36, collectively indicating the disrupted lipid metabolism [3–5]. This evidence concerns the gene CD36 and fetal growth restriction.