CXCL10 was linked to Allograft Rejection, Butanoate Metabolism, Glyoxylate and Dicarboxylate Metabolism, Graft-versus-host Disease, Nitrogen Metabolism, and Systemic Lupus Erythematosus pathways (Figure 6E), while LAT2 was associated with Asthma, Glycine, Serine and Threonine Metabolism, Glyoxylate and Dicarboxylate Metabolism, Nitrogen Metabolism, Primary Immunodeficiency, and Selenocompound Metabolism pathways (Figure 6F). This evidence concerns the gene CXCL10 and inborn error of immunity.