Moreover, the gene expression profile indicated that MMP11 was primarily localized within the F01 cell population (Figure 5G), while this cell population exhibited characteristics of angiogenesis (CXCL14, POSTN, SFRP2, CXCL12) and a matrix-immune suppressive phenotype (COL1A1, CTHRC1, CCL11) (Figure 5G); these suggested a potential association between F01 and PCa progression. The gene discussed is CCL11; the disease is posterior cortical atrophy.