With the low levels of IFNγ detected in the human and mouse artery tissue scRNAseq data sets we cannot rule out that IFNγ may not be the, or not the only, cytokine that induces a STAT1 pattern of gene expression in atherosclerosis lesions in as much that other cytokines can also induce GAF and/or ISGF3 activity in myeloid cells including type I interferons in some settings, IL-6, and IL-27. This evidence concerns the gene FGF9 and atherosclerosis.