The gel slowly released basic FGF, which improved the local microenvironment, activated endogenous NSCs and recruited these cells to migrate to the hemidiaphragm and the stroke cavity, and enhanced angiogenesis in the hemidiaphragm and the stroke cavity, and the results showed that bFGF-chitosan gel strongly stimulated the vascular network in and around the stroke cavity. The gene discussed is FGF2; the disease is stroke disorder.