Interleukin 10, interleukin 33, and interleukin 34 (IL-10, IL33, and IL34), and induced the differentiation of Tregs from T cells, which greatly ameliorated the inflammation at the site of injury and promoted the repair of MI-injured myocardial tissue (Zhu et al., 2022). This evidence concerns the gene IL10 and myocardial infarction.