The role of inflammation in acute ischemic stroke (AIS) has become increasingly recognised, with mounting evidence associating elevated inflammatory biomarkers—such as C-reactive protein (CRP), interleukins (e.g., IL-6), and tumor necrosis factor-alpha (TNF-α)-with more severe strokes and poorer functional recovery (13, 14). This evidence concerns the gene CRP and androgen insensitivity syndrome.