This was further supported by the system's ability to reduce the release of pro‐inflammatory cytokines, including TNF‐α and IL‐6 (Figure 6D), leading to a 60% improvement in survival in an in vivo mouse model of LPS‐induced endotoxemia and E. coli‐induced sepsis, as demonstrated in Figure 6E. The gene discussed is TNF; the disease is serum lipopolysaccharide activity.