Among these, SF3B1, a well‐characterized component of the U2 snRNP complex, has been reported to recognize 3′ splice sites and promote EC progression by regulating KSR2 mRNA maturation.[30, 31] SF3B1 mutations are also frequently observed in uveal melanoma, with hotspot mutations detected in ≈20% of cases.[31]. The gene discussed is SF3B1; the disease is uveal melanoma.