Our analysis of human ILC samples further support a paracrine IL-6 signaling pathway in ILC where there is a strong positive correlation between IL6 and the stromal content of tumors, and a correlation between stromal IL6 and nuclear STAT3 within the tumor compartment and limited IL6 expression in the epithelial compartments of both mILC and human ILC tumors in scRNA-Seq datasets. This evidence concerns the gene STAT3 and neoplasm.