EPAS1 and neoplasm: Differences in ARNT-dependent and BMAL1-dependent HIF2α regulation of endogenous target genes and in the sensitivity of ARNT-HIF2α and BMAL1-HIF2α heterodimers to disruption by HIF2α antagonist drugs suggest that the relative expression of ARNT and BMAL1 could influence the tumor growth suppression response to HIF2α antagonists.