Taken together, these results show that, in our mouse models of TNBC, the CDDP-Eri combination did not result in antitumor synergy but did appear to be immunologically additive, inducing a global inflammatory and immune tumor response, accompanied by intratumoral PD-L1 expression, as well as intratumoral recruitment of activated, cytotoxic, proliferative, and polyfunctional CTLs. Here, CD274 is linked to neoplasm.