A recent publication reported that individuals with post-infectious IBS were found to have increased numbers of neutrophils and T helper 17 cells, higher interleukin 7 levels, and widened epithelial cell gaps compared to individuals with non-post-infectious IBS [34], suggesting gastrointestinal infection may lead to lasting changes in the immune and structural profile of the gut, with subsequent symptoms. Here, IL7 is linked to irritable bowel syndrome.