TGFB1 and infection: This expression pattern resembles that of other growth factors, such as EGF, PDGF, and TGF‐β, which contribute to skin homeostasis.[49, 50] Previous studies have established that wound healing involves a coordinated response of multiple cell types, with leukocytes initially infiltrating the wound to combat infection and prevent blood loss, followed by fibroblasts proliferating to deposit collagen and regenerate tissue.[1] Our study aligns with this paradigm, demonstrating that gcFGF8a enhances leukocyte recruitment at day 3 post‐injury and collagen deposition at day 7.