WT1 and myocardial infarction: In the acute phase (48-72 hours) after MI, we did not detect significant differences in heart- to-body weight ratios, histology, cardiomyocyte diameters, and infarct sizes (Figure S5A-I) in VE-CreERT2;Wt1lox/lox+Tamoxifen animals compared to the respective controls while CD31 immunostaining revealed a significant reduction of left ventricular vessel density in animals with endothelial-specific knockout of WT1 after MI (Figure S5J-L).