,63 Extensive studies have revealed that m6A modification is involved in cancer hallmarks by regulating tumor-related gene expression at post-transcriptional stages.64, 65, 66 IGF2BP1, as a vital m6A reader, specifically recognizes and binds to m6A methylation mRNAs, leading to the increase of their stability and translation.34 The gene discussed is IGF2BP1; the disease is cancer.