Post-transplant hypertriglyceridaemia can be induced by genetic predisposition, alcohol consumption, high carbohydrate intake, diabetes, overweight and obesity, proteinuria, renal failure and IS therapy [mostly glucocorticosteroids (GCS), mammalian target of rapamycin (mTOR) and calcineurin-inhibitors (CNIs)]. This evidence concerns the gene MTOR and kidney failure.