According to Lee et al. [92], it can cleave and activate caspases 3/9 and poly (ADP-ribose) polymerase (PARP), downregulate the expression of X-linked inhibitor of apoptosis protein (XIAP), block the cell cycle in the G2/M phase, and induce oxidative stress to inhibit breast cancer proliferation. The gene discussed is XIAP; the disease is breast carcinoma.