With additional evidence from previous literature reporting enhanced anti-tumor effects of dual inhibition (inhibition of Bcl-2 and FGFR in endometrial cancer; and of Bcl-2 and mTOR in renal cell carcinoma) (Packer et al. 2019; Nayman et al. 2019), we hypothesize that both signaling pathways may be able to implement resistance to BCL2 inhibition in OVCA and may contain reasonable combination targets with BCL2i. The gene discussed is BCL2; the disease is neoplasm.