IFN-γ was the critical mediator in this dormant tumor phenotype as neutralizing IFN-γ antibodies, but not antibodies to TNF-α, decreased CVCPos and increased Ki-67+ D2A1 cells in a coculture of CD103+DCs and CD8+ T cells from Tgfbr2MyeKO mice with CVC labeled D2A1 tumor cells (Supple fig. This evidence concerns the gene MKI67 and neoplasm.