PLA2R1 and membranous glomerulonephritis: A potential cause of this increased risk is linked to particulate matter 2.5 (PM2.5), a type of air pollution consisting of extremely small particles that can be inhaled [28] and subsequently elicits oxidative stress and upregulates PLA2R expression in the lungs, resulting in the pathogenesis of membranous nephropathy through extracellular vesicles [28].