CD36 and myocardial infarction: In addition, a C/T substitution in exon 4 (rs75326924) in the CD36 gene has been reported to be associated with a significant reduction in myocardial LCFAs uptake in patients with angina pectoris, myocardial infarction, hypertrophic cardiomyopathy, dilated cardiomyopathy, hypertension, aortic stenosis, and mitral valve disease, but only in the Japanese population [38].