Moreover, a recent study by Khan et al. describes variant c.541G>A p.G181R in MYH7 (affecting the same amino-acid as in case of our variant p.G181V) found in pediatric patients with dilated cardiomyopathy with left ventricular noncompaction features (DCM-LVNC) and classified as pathogenic/likely pathogenic (P/LP) [23]. This evidence concerns the gene MYH7 and left ventricular noncompaction.