A recent study found that GLI1 transcriptionally regulates FANCD2 expression, a tumor suppressor gene, and a key member of the FA-BRCA and the homologous recombination (HR) pathways, implicating the importance of GLI1-dependent expression of FANCD2 genes in tumorigenesis and chemoresistance in OC [44]. This evidence concerns the gene FANCD2 and Friedreich ataxia.