In breast cancer, it inhibits tumor growth and metastasis, enhancing survival by regulating mitochondrial fission via dynamin-related protein 1 (Drp-1) and promoting autophagy through autophagy-related protein 2 (ATG2), making it a favorable prognostic marker—particularly valuable for triple-negative breast cancer subtypes [36,37,38]. This evidence concerns the gene DNM1L and triple-negative breast carcinoma.