NFKB1 and disseminated peritoneal leiomyomatosis: In cases such as this, where the histology reveals polymorphic LPD with Hodgkin-like features, it is plausible that EBV-driven activation of oncogenic pathways, including NF-κB and JAK/STAT signaling, contributes to the proliferation and survival of malignant or pre-malignant B-cell clones [16,17].