It is well known that food protein-induced sensitization can manifest by high TNF-α expression in the small intestine and that TNF-α increases intestinal permeability in patients with inflammation [50]; IL-6 levels are correlated with the severity of inflammatory changes in the intestine [51]; and IL-10 is a key anti-inflammatory cytokine that ensures the protection of the host from overreactions to pathogens and microbiota while playing an important role in the treatment of enteritis, autoimmunity, cancer, and homeostasis in other environments [52]. The gene discussed is TNF; the disease is Autoimmunity.