TGFB1 and abdominal aortic aneurysm: In an experimental study, Dai et al. showed that active TGFB1 overexpression through endovascular monogenic delivery or treatment with recombinant TGF-β1 protein leads to a reduction in proteolytic activity, mitigates wall degradation, facilitates tissue regeneration on the luminal surface, and contributes to the stabilization of abdominal aortic aneurysms [47].