Given that miR-193-5p promotes autophagy and inhibits SRSF10 (which in turn impairs autophagy), and that its expression is suppressed by lncRNA GAS5 (a molecule that contributes to atherosclerosis and impairs autophagy), it is suggested that in normal endothelial cells, miR-193-5p is maintained at a level that supports healthy endothelial cell autophagy by regulating the expression of SRSF10 [85]. Here, SRSF10 is linked to atherosclerosis.