The expression of PLK1, a key regulator of cell cycle progression and a marker of cellular proliferation [18,19,20,21,22,23], was inversely correlated with the vector-prone increase in DAPK1 re-expression (Figure 3C), supporting an anti-proliferative role of DAPK1 in ovarian cancer. The gene discussed is DAPK1; the disease is ovarian carcinoma.