These oncogenes can form through changes such as chromosomal translocation (e.g., chromosomal translocation of the MYC oncogene in Burkitt’s lymphoma), point mutation (e.g., point mutation at codon 12 of the RAS oncogene), and gene amplification (e.g., amplification of c-MYC in neuroblastoma) [5]. The gene discussed is MYC; the disease is Burkitt lymphoma.