These genes span key functional categories, including glutathione synthesis (as Gclc), mitochondrial detoxification (as Gpx6), redox cycling (as NQO1), selenium transport (as SEPP1), immune signaling (as CCL5), and DNA recombination (as Rag2), underscoring the multifaceted disruption of redox regulation in the MetS kidney. This evidence concerns the gene CCL5 and metabolic syndrome.