MYOD1 mutations are associated with poor prognosis in rhabdomyosarcoma [30]; DYRK1A is a conserved kinase implicated in the pathogenesis of several diseases, including cancer and neurodegeneration [31]; DICER1, a key RNA‐processing endonuclease, is linked with hereditary tumor susceptibility syndromes [32] and INTS6 regulates gene expression through premature transcriptional termination and functions a context‐dependent tumor regulator [33, 34]. Here, DYRK1A is linked to rhabdomyosarcoma.