This suppression reduces the production of key cytokines implicated in psoriasis, such as TNF-α, IL-17, IL-23, and IFN-γ, thereby attenuating keratinocyte hyperproliferation and immune cell infiltration (Li et al. 2018; Vujic et al. 2018; Tsentemeidou et al. 2022). Here, IL17A is linked to psoriasis.