More recently, the overexpression of DNA J homolog subfamily B member 9 (DNAJB9) has been identified in both idiopathic FGN and FGN associated with other conditions, including systemic lupus erythematosus (SLE), rheumatoid arthritis, hepatitis C virus infection, monoclonal gammopathy, diabetes mellitus, and chronic infections [7,8,9]. The gene discussed is DNAJB9; the disease is systemic lupus erythematosus.